Study: Cell Phones Don’t Cause Cancer in Kids
Cell phones don’t increase kids’ cancer risk, a new study suggests. Researchers compared the cell phone habits of nearly 1,000 children and adolescents ages 7 to 19 in Western Europe, including 352 who had been diagnosed with a brain tumor between 2004 and 2008, and 646 who were healthy. They found that regular cell phone users were no more likely to have developed a brain tumor than were never-users, according to findings published Wednesday in the Journal of the National Cancer Institute. In fact, similar percentages of children with and without brain tumors—75 percent and 72 percent, respectively—had used a cell phone at least 20 times before diagnosis, suggesting there is no "causal association between the use of mobile phones and brain tumors," the researchers wrote. They did note that most study participants had only been using cell phones for about four years, which may not be long enough to accurately determine cancer risk, since the disease takes a long time to develop, Reuters reports. In an accompanying editorial, scientists recommended continuing to monitor brain tumor rates among children and other cell phone users.
What Causes Cancer? 7 Strange Cancer Claims Explained
Bras, deodorant, and mouthwash—just a few of the everyday products that have been linked to cancer at some point during the past several decades. Preposterous? Not at the time, and new suspects have been added to the list. Here’s the real story behind a roll call of ordinary household items that have come under scrutiny:
1. Artificial Sweeteners. The link: Calorie watchers scored a win when diet sodas were introduced in the early 1950s. Then lab studies suggested that the sweetener cyclamate caused bladder cancer in rats, and the U.S. Food and Drug Administration banned its use. Then saccharin, the replacement of choice, was also shown to cause tumors in rats. Although saccharin was never banned, all products containing the sweetener were required to carry a cancer warning on their packaging.
The reality: No evidence has since emerged that either cyclamate, which is used in other countries, or saccharin causes cancer in humans, according to the National Cancer Institute. Although cyclamate is still banned, in 2000 saccharin was taken off the government’s list of possible carcinogens and saccharin products shed the warning label. The sweetener aspartame has come under suspicion, but scientists have found no increased risk of cancer in humans. [Read more: What Causes Cancer? 7 Strange Cancer Claims Explained.]
Cancer Prevention: Rethink Your Diet as Well as Your Smoking
If everyone were to quit smoking , nearly 450,000 fewer Americans would die annually from smoking-related diseases, U.S. News reported in 2010. Yet even with all the smoking bans across the country, one in five Americans still lights up regularly—a rate that’s plateaued since 2005 after four decades of decline, according to a report issued last year by the Centers for Disease Control and Prevention. While the smoking-lung cancer connection is an old story, every week, it seems, another headline tells you what you should or shouldn’t eat to avert the "big C". Eat a colorful array of fruits and vegetables to ward off lung cancer, says one recent study; avoid soft drinks if you don’t want to die of pancreatic cancer, warns another. Wine is good for your heart, but may increase your risk of breast cancer, others suggest.
And who wouldn’t be willing to give up the Diet Coke or chardonnay to sidestep the disease we fear most? It you took action based on research published in the Annals of Internal Medicine, you might find yourself forgoing hamburgers and chicken wings and embracing soy burgers, tofu, and peanut butter instead. That study found that an Atkins-style diet that emphasized vegetable protein over animal protein lowered the risk of cancer. But all things considered, how much do dietary changes really matter in terms of cancer prevention? "Right after smoking, diet ranks right up there as the No. 2 modifiable risk factor," says Demetrius Albanes, a senior investigator and medical epidemiologist at the National Cancer Institute. "Twenty-five percent of cancers can be related back to eating practices." Does that mean we really need to avoid soft drinks if we don’t want to get pancreatic cancer, as was suggested by a study in the journal Cancer Epidemiology, Biomarkers & Prevention? "I don’t think we can say that," Albanes says. "That particular study from Singapore didn’t control very well for smoking, which is an important risk factor for pancreatic cancer." In other words, it could be that those who down daily six-packs of Fanta are also more likely to light up. [Read more: Cancer Prevention: Rethink Your Diet as Well as Your Smoking.]
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CDC: Spike in Pregnancy-Related Strokes
The stroke rate among pregnant women and new moms is increasing at an "alarming" rate, the federal government warned Thursday. Researchers from the Centers for Disease Control and Prevention analyzed hospital-discharge data, and found that between 1994-5 and 2006-07, the rate of stroke hospitalizations rose 47 percent for expectant mothers and 83 percent for women who had recently given birth. Although the overall incidence remains low—0.22 stroke hospitalizations per 1,000 expectant and new moms—the increase is worrisome, say the study authors. The most likely explanation is that pregnant women are increasingly likely to have other risk factors for stroke, such as obesity, high blood pressure, and diabetes. "That is a very, very alarm-raising statistic that we need to take extremely seriously," Olajide Williams, a neurologist at Columbia University and an American Stroke Association spokesman, told USA Today. "We need to be more aggressive in screening these women for these risk factors." The findings were published in Stroke: Journal of the American Heart Association.
Stroke: 7 Signs You Could Be at Risk of a Brain Attack
Stroke can hit like a deadly lightning bolt. And if the victim survives, the aftermath can be debilitating—affecting functioning from movement to speech. While stroke is the third-leading cause of death and the leading cause of adult disability in the United States, it trails behind other major diseases in awareness and recognition of symptoms. Being informed, however, can protect you from suffering either an ischemic stroke, caused by a blood clot and the most common form of stroke, or the less common hemorrhagic stroke, caused by bleeding in the brain. Know the factors that may be putting you at risk:
Uncontrolled high blood pressure. As for all cardiovascular disease, high blood pressure is a major risk factor for stroke. The American Heart Association estimates that only 45 percent of people with high blood pressure actually have it under control, U.S. News reported in 2009. Female stroke victims, in particular, tend to have uncontrolled blood pressure, and in general, women who suffer strokes don’t seem to be treated as aggressively as men. High blood pressure doesn’t have any outward telltale signs, so getting it measured by your healthcare provider is essential to determine if you should make lifestyle changes or take medications to bring it down.
Smoking. Puffing on cigarettes is associated with a host of ills. An increased risk of stroke is one of them. When compared to nonsmokers, smokers have double the risk of ischemic stroke. Heavy smokers face an even greater risk: A study of women ages 15 to 49 published in the journal Stroke found stroke risk was proportional to the number of cigarettes smoked per day. The women who smoked two or more packs a day had nine times the risk of stroke of a nonsmoker. And a study in Neurology found that smokers with a family history of brain aneurysm, abnormal bulging of an artery in the brain, are six times as likely to have a subarachnoid hemorrhage, a kind of stroke caused by a bleed between the brain and the tissue that covers it. These types of stroke are deadly nearly 40 percent of the time. [Read more: Stroke: 7 Signs You Could Be at Risk of a Brain Attack.]
5 Symptoms You Need to Know to Recognize a Stroke Immediately
Minimizing the time between the onset of a stroke and the start of stroke treatment is critical for surviving the brain attack and minimizing the resulting brain injury. The key is to immediately get to the emergency room for a brain scan to detect which type of stroke has hit. If it’s ischemic—caused by a blood clot—the best treatment is a clot-dissolving drug called tissue plasminogen activator, or TPA, and the quicker the treatment, the less the disabling damage. Most hospitals will treat stroke patients with TPA only if the medicine can be injected within three hours of the appearance of symptoms, which is why getting to the hospital is such an urgent matter. One study found, however, that TPA can be safe and effective up to 4½ hours after a stroke. Treatment for hemorrhagic stroke, caused by a bleeding vessel in the brain, involves lowering blood pressure and reducing swelling in the brain, U.S. News reported in 2009.
Stroke can present itself with a range of symptoms, but the consistent factor is that they come on suddenly. Call 911 immediately if you, or someone you’re with, experience any of the following:
Numbness or weakness, particularly on one side of the body. This can be in the face, an arm, or a leg. If someone you’re with appears to be experiencing this, ask the person to smile, lift both arms, or move both legs, the National Stroke Association recommends. If one side of the body doesn’t respond, it may be a sign of stroke. [Read more: 5 Symptoms You Need to Know to Recognize a Stroke Immediately.]
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Doctors do the best they can to keep patients healthy and out of the hospital. Sometimes, though, hospitalization is necessary despite the best possible care. At the first private practice I joined after residency training, my colleagues and I admitted patients to a local hospital and took turns caring for those who needed inpatient treatment. This system ensured we’d have easy access to their previous medical records and often know them on a personal level. Arranging office appointments after discharge was almost never a problem, and we were guaranteed knowledge of what had happened to our patients in the hospital.
That’s all changed. The way patients receive hospital care has transformed radically in the past 15 years. Many primary care physicians, pressured to take on more patients and exhausted from being on call too many nights, have stopped seeing their patients through a hospital stay. Instead, they now rely on "hospitalists," a relatively new breed of specialists whose exclusive responsibility is to care for hospitalized patients.
There are potential advantages to being treated by a hospitalist rather than a family doctor. Because hospitalists spend all their time on the wards rather than trying to juggle obligations to hospital and office patients, they’re usually easier to reach with questions or concerns. Also, hospitalists may be more up-to-date on the latest medical research on inpatient treatments. These advantages should theoretically translate into better care and shorter stays for hospital patients. And they do, according to a 2007 study published in the New England Journal of Medicine; researchers found that patients cared for by hospitalists indeed had shorter hospital stays and lower medical costs than those cared for by primary care physicians.
But as hospitalists are replacing family doctors on hospital wards, concern is mounting that poor communication between hospital and office physicians could lead to worse health outcomes after discharge. I personally know the frustration of seeing a patient in the office after a recent hospitalization having not received critical information about what medication changes were made, what procedures he underwent, or what tests are needed to monitor his condition. Patients whose doctors don’t have access to complete information during follow-up visits may be more likely to end up in the emergency room or be hospitalized yet again. (Same goes for patients who don’t schedule follow-up visits at all.)
A study published Monday in the Annals of Internal Medicine seems to confirm these fears. In a nationally representative sample of Medicare patients admitted to hospitals between 2001 and 2006, those who were cared for by hospitalists had slightly shorter average hospital stays and slightly lower hospital bills than those cared for by primary care physicians. However, in the 30 days after discharge, hospitalist patients were more likely to be readmitted or land in the emergency room. One possible explanation: poor communication, since hospitalist patients were significantly less likely to follow up with their primary care physicians after discharge.
To improve the quality of "care transitions" between hospitalists and family doctors, some health systems have devised programs to ensure patients get the recommended follow-up care. Two studies published in last week’s Archives of Internal Medicine evaluated two such programs. In one study, seniors who’d been hospitalized for heart failure at Baylor Medical Center in Garland, Texas received several home visits by specially trained nurses between three days and three months after discharge. Those enrolled in the nurse-visit program were only half as likely as past heart failure patients to be readmitted within 30 days. In another study, patients at six Rhode Island hospitals were assigned health coaches (nurses or social workers) who visited them once in the hospital, once at home, and telephoned them twice to encourage follow-up with primary care physicians and ask about any worrisome signs or symptoms. Patients in that program were nearly 40 percent less likely to be readmitted within 30 days than patients who received no health coaching.
Because it’s impossible to predict whether you or a loved one will need to be hospitalized, it’s important to understand your doctor’s policies for patients who require hospital care. You can start by asking these 4 questions:
Given all of the changes that have taken place in medicine, it’s unlikely that we will return to the "old days" when the same doctors were responsible for caring for their patients both in and out of the hospital. Consequently, patients need to be proactive to be sure that they receive the best post-hospital care. Being hospitalized is always stressful, but knowing that your follow-up care won’t fall through the cracks may give you peace of mind.
Tainted Turkey Is Likely Source of Latest Salmonella Outbreak
An antibiotic-resistant strain of salmonella has made its way into at least 26 states, killing one person and sickening 77. On Wednesday, U.S. health officials were investigating the source of the outbreak, which they believe is tied to tainted ground turkey. The probe so far points to a single facility—the location of which is being withheld—that has produced three of four salmonella-infected ground turkey samples taken from four stores. The salmonella strain involved worries officials because it is resistant to common antibiotics, raising the risk of hospitalization and treatment failure, the CDC says. Symptoms of salmonella infection include: diarrhea, fever, and abdominal cramps. Although most people recover without treatment, the infection can be deadly, especially if treatment fails and it’s allowed to spread from the intestines to the bloodstream. Older adults, infants, and those with weakened immune systems are especially vulnerable.
How to Reduce Your Risk of Salmonella Infection
Salmonella is scary, but you can take steps to prevent infection with basic precautions, U.S. News reported in 2009.
1. Eat well-cooked food. In recent years, some of the nastiest food poisoning outbreaks have been caused by fresh fruit and veggies—canteloupes and jalapeño peppers in 2008, and spinach in 2006. Washing doesn’t solve the problem, since pathogens can get inside the nooks and crannies of a cantaloupe rind, for example. There’s also evidence that leafy greens and tomatoes can slurp bacteria into their cells along with water, either in the field or during processing. As Doug Powell, director of the International Food Safety Network at Kansas State University, says: "Washing’s not enough. You gotta cook it." If you’re worried, sautéed spinach is a better bet than salad. Cooking meats thoroughly eliminates pathogens common in chicken and ground beef. [Read more: New Salmonella Outbreak: Here's How to Reduce Your Risk]
Would Your Kitchen Pass a Restaurant Inspection?
If a restaurant inspector barged into your kitchen tomorrow, would it pass the test—or would he threaten to shut you down? Clipboard in hand, he’d check the temperature inside the refrigerator. Warmer than 40 degrees? Violation. Raw meat stored above ready-to-eat food? More points off. Same goes for dirty, cracked eggs, and swollen, leaking, or rusted cans of food. And don’t even think about smoking while you’re cooking.
At least one in seven home kitchens would flunk a restaurant-type health inspection, a recent study by the Los Angeles County Department of Public Health suggests, and only three out of five would earn an A or B, U.S. News reported in 2010. Since food consumed at home is the source of roughly half of the nation’s annual 76 million cases of food-borne illnesses, that’s worrisome. "Sometimes we get a little sloppy in our own kitchens," says Joan Salge Blake, a registered dietitian and nutrition professor at Boston University. "Whether you’re bringing raw food into your home to prepare or leftovers from a restaurant, you have to do your part to help reduce the risk of coming down with a food-borne illness." [Read more: Would Your Kitchen Pass a Restaurant Inspection?]
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Marie Gilbert, University of Alaska, Fairbanks
Hibernation is an essential survival strategy for some animals and scientists have long thought it could also hold promise for human survival. But how hibernation works is largely unknown. Scientists at the University of Alaska Fairbanks have successfully induced hibernation at will, showing how the process is initiated. Their research is published in the July 26 issue of The Journal of Neuroscience.
A hibernating animal has a reduced heart rate and blood flow similar to a person in cardiac arrest, yet the hibernator doesn’t suffer the brain damage that can occur in people.
“Understanding the neuroprotective qualities of hibernating animals may lead to development of a drug or therapy to save people’s lives after a stroke or heart attack,” said Kelly Drew, senior author and UAF professor of chemistry and biochemistry in the Institute of Arctic Biology.
Hibernating animals survive by severely reducing their metabolism, a condition called torpor, in which oxygen consumption can fall to as low as one percent of resting metabolic rate and core body temperature to near or below freezing temperatures.
Arctic ground squirrels, like all animals and people, produce a molecule called adenosine that slows nerve cell activity.
“When a squirrel begins to hibernate and when you feel drowsy it’s because adenosine molecules have attached themselves to receptors in your brain,” said Tulasi Jinka, lead author and a postdoctoral fellow in Drew’s lab.
The receptors can be regulated by a simple cup of coffee. A caffeine molecule is similar enough in structure to adenosine that it binds to the receptors and effectively stops or reverses the onset of drowsiness. Jinka and Drew wanted to know what substances trip the squirrels’ switch to start to hibernate.
“We devised an experiment in which non-hibernating arctic ground squirrels were given a substance that stimulated adenosine receptors in their brains. We expected the substance to induce hibernation,” Drew said. “We also gave a substance similar to caffeine to arouse hibernating ground squirrels.”
The non-hibernating squirrels were tested three times during one year. They were tested during the summer when they were not hibernating, again early in their hibernation season and a third time midway through the hibernation season. If animals were hibernating before the test, Jinka woke them up to see if the substance would cause them to go back into hibernation. To ensure that his expectations did not influence the results he delivered a placebo in the same manner as the drug and did not know which solution contained the active substance when he conducted the experiments.
Torpor was induced in all six of the squirrels awoken during mid-hibernation season, but in only two of the six from the early hibernation season group and in none during the summer season. The caffeine-like substance reversed torpor in all of the hibernating squirrels.
“We show for the first time that activation of the adenosine receptors is sufficient to induce torpor in arctic ground squirrels during their hibernation season,” Jinka said, who conducted this experiment while he was a graduate student.
What Jinka and Drew don’t yet know is how season causes the receptors to become increasingly sensitive to adenosine as the time of hibernation progresses.
Jinka and Drew are expanding their adenosine research to rats, which more closely resemble the physiology of humans.
“Rats allow us to move toward being able to apply this research to humans,” Jinka said.
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By Joel N. Shurkin, ISNS Contributor
(ISNS)—Life for elephant seals is nasty and brutish—but their sex lives are legendary.
Only the biggest and most brutish males are believed to reproduce. During the annual breeding season, huge males, called beachmasters, rule harems of much smaller females. The beachmaster drives off all male competitors until another big, aggressive male comes along and dethrones him. It is believed to be one of nature’s purest examples of polygyny.
In this case, evolution favors the strongest and biggest.
It turns out those helpless females may not be so helpless, at least in one colony. Large numbers of female Southern elephant seals at the edge of Antarctica are ignoring the polygyny system in favor of sex in the high seas.
But evolution may also favor the wisest.
Researchers from Australia and South Africa have found that large numbers of female seals skip the yearly breeding interval at Marion Island, approximately located between the two continents of Africa and Antarctica. When they finally do show up, they are pregnant, and the beachmaster had nothing to do with it. Almost three-fourths of the males skip the violence and frustration as well.
955 miles off the African coast, Marion Island is a wildlife preserve full of seals, penguins and seabirds that is administered by South Africa. According to Nico de Bruyn, a marine mammal ecologist at the University of Pretoria, the seals come to land twice a year: once to molt, the other to reproduce. Both tasks are much easier to do on solid land than in the tossing ocean.
Male bull elephant seals are enormous, some measuring 16 feet long and weighing more than 3 short tons.They have a proboscis, or trunk, almost a foot long, hence the elephant in their name. They are loud, smelly and mean-tempered.
Female elephant seals are one-fourth the weight and up to 6 feet shorter than males.
The seals employ delayed implantation, meaning the fertilized egg does not implant itself and begin development until the timing is exactly correct for the pup to be born on land, a 12-month gestation period including the delay. On Marion Island, that is in October, de Bruyn said. All the pups are born within days of each other.
Females nurse the young for only about three weeks and are then ready to reproduce again. The bulls, alpha males, are so large many females and pups are killed when an amorous or bellicose bull lands on them. On the crowded beach there is no place to escape.
But the researchers, reporting in Animal Behaviour, have found that as many as half the females refuse to play along, staying in the ocean and mating with whom they chose rather than joining the chaos on the beach.
"For a male, even if he is huge in comparison to the female—which they are—coercing a female is so much more difficult in the water because she has more options," de Bruyn said.
The researchers began their work of marking and capturing animals because of concern about a dramatic decline in Southern elephant seals noted in the 1980s. The population at Marion Island was down 80 percent for reasons unknown, although de Bruyn said that it has stabilized now and is even showing a slight increase.
De Bruyn does not think this evasion of is new behavior.
Oddly, that behavior is not seen in other elephant seal colonies. Daniel Costa, professor of ecology and evolutionary biology at the University of California, Santa Cruz, studies the Northern elephant seals that breed at Ano Nuevo on the California coast.He doesn’t think that behavior is common.
"We have no evidence that it occurs in Northern elephant seals.I’m not even sure how prevalent the observation is for Southern elephant seals," Costa said. "We know that our females do not hang out in the water. I can say this because we track them onto the beach from sea, or at least the 300-plus females we have tracked don’t spend a lot of time offshore. They just come in and land on the beach."
And then end up in some beachmaster’s harem.
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When people recognize voices, part of what helps make voice recognition accurate is noticing how people pronounce words differently. But individuals with dyslexia don’t experience this familiar language advantage, say researchers.
The likely reason: "phonological impairment."
Tyler Perrachione with the Massachusetts Institute of Technology explains, "Even though all people who speak a language use the same words, they say those words just a little bit differently from one another–what is called ‘phonetics’ in linguistics."
Phonetics is concerned with the physical properties of speech. Listeners are sensitive to phonetic differences as part of what makes a person’s voice unique. But individuals with dyslexia have trouble recognizing these phonetic differences, whether a person is speaking a familiar language or a foreign one, Perrachione says.
As a Ph.D. candidate in Neuroscience at MIT, Perrachione recently examined the impacts of phonological impairment through experiments funded by the National Science Foundation’s Directorate for Education and Human Resources.
He and colleague Stephanie Del Tufo as well as Perrachione’s MIT research advisor John Gabrieli hypothesized that if voice recognition by human listeners relies on phonological knowledge, then listeners with dyslexia would be impaired when identifying voices speaking their native language as compared to listeners without dyslexia.
They also theorized that listeners with dyslexia hearing a foreign language would be no more impaired in voice recognition than listeners without dyslexia, because both groups would lack specific familiarity with how the foreign language was supposed to sound.
The journal Science reports their findings online today in an article titled "Human voice recognition depends on language ability."
For their research study, the MIT scientists trained individuals with and without dyslexia to recognize the voices of people speaking either the listeners’ native language of English or an unfamiliar foreign language, Mandarin Chinese. In each language, participants learned to associate five talkers’ voices with unique cartoon avatars and were subsequently tested on their ability to correctly identify those voices.
The listeners were either typically-developing readers or individuals who experienced reading difficulties and dyslexia growing up.
The neuroscientists found individuals with dyslexia were significantly worse at being able to consistently recognize the voices of the English speakers. They were about the same as listeners without dyslexia at recognizing the Chinese voices; both groups were very poor at recognizing voices speaking an unfamiliar language.
"It is remarkable that individuals with dyslexia are no better able to identify voices speaking a familiar language than a foreign one," says Perrachione. "It is also very interesting that the reason for this is that they are less accurate at voice recognition than individuals who don’t have dyslexia."
The result reaffirms the theory that the underlying deficit in dyslexia isn’t about the act of reading per se, but instead involves difficulty with how sounds of spoken language are heard and processed in the dyslexic brain.
Contemporary theories of dyslexia often propose a "phonological deficit" as the reason some people struggle to translate written images into meaningful language. The idea is that individuals with dyslexia tend to do poorly on tests that ask them to decode words using conventional phonetic rules, thereby resulting in reading delays because of difficulties connecting language sounds to letters.
What theories of dyslexia have not been able to convincingly explain, say the researchers, is why there is no evident difficulty in the ability to perceive and produce speech among people with dyslexia. This is especially curious if the ability to recognize phonological sounds is impaired.
"Our results are the first to explicitly link impairment in reading ability to impairment in ecologically processing spoken language," says Perrachione. "The results suggest that the source of a phonological deficit might be in dyslexic individuals’ difficulties learning the consistent properties of speech sounds as spoken by an individual talker."
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